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by Mike Rust
Why have the bacterial signaling systems based on histidine and aspartate phosphorylation been abandoned (with some exceptions) in favor of serine/threonine and tyrosine phosphorylation in eukaryotes? Is this just a "frozen accident" or is there a deep biochemical reason?
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interesting question? Maybe this is due to relative stability of the phosphate-hydroxyl bond?
Posted by: Dave_bridges | November 18, 2010 at 12:36 PM